Early Neuroaxonal Damage in Neurologic Disorders Associated With GAD65 Antibodies
2024-01 | journal article
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Cite this publication
Early Neuroaxonal Damage in Neurologic Disorders Associated With GAD65 Antibodies
Eisenhut, K.; Faber, J.; Engels, D.; Gerhards, R.; Lewerenz, J.; Doppler, K. & Sommer, C. et al. (2024)
Neurology: Neuroimmunology & Neuroinflammation, 11(1). DOI: https://doi.org/10.1212/NXI.0000000000200176
Documents & Media
Details
- Authors
- Eisenhut, Katharina; Faber, Jennifer; Engels, Daniel; Gerhards, Ramona; Lewerenz, Jan; Doppler, Kathrin; Sommer, Claudia; Markewitz, Robert; Falk, Kim K.; Rössling, Rosa; Pruess, Harald; Finke, Carsten; Wickel, Jonathan; Geis, Christian; Ratuszny, Dominica; Pfeffer, Lena K.; Bittner, Stefan; Piepgras, Johannes; Kraft, Andrea; Klausewitz, Jaqueline; Nuscher, Brigitte; Kümpfel, Tania; Thaler, Franziska S.
- Abstract
- Neurodegeneration is considered a relevant pathophysiologic feature in neurologic disorders associated with antibodies against glutamic acid decarboxylase 65 (GAD65). In this study, we investigate surrogates of neuroaxonal damage in relation to disease duration and clinical presentation.
- Issue Date
- January-2024
- Journal
- Neurology: Neuroimmunology & Neuroinflammation
- Project
- TRR 274: Checkpoints of Central Nervous System Recovery
TRR 274 | C02: In vivo detection and targeting of calcium clearance and axonal membrane repair after acute CNS insults - Working Group
- RG Kerschensteiner (Neuroimmune Interactions)
- ISSN
- 2332-7812
- Language
- English
