Nuclear NFATc1 Causes Development of Fms-like Receptor Tyrosine Kinase 3 Receptor (FLT3) Internal Tandem Duplication Mutation-Positive (FLT3-ITD) Acute Myeloid Leukemia (AML) and Mediates Drug Resistance

2016 | journal article. A publication with affiliation to the University of Göttingen.

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​Nuclear NFATc1 Causes Development of Fms-like Receptor Tyrosine Kinase 3 Receptor (FLT3) Internal Tandem Duplication Mutation-Positive (FLT3-ITD) Acute Myeloid Leukemia (AML) and Mediates Drug Resistance​
Solovey, M. A.; Wang, Y.; Gattenloehner, S.; Hessmann, E. ; Metzelder, S.; Michel, C. & Ellenrieder, V.  et al.​ (2016) 
Blood, 128(22) pp. 1722​-1722​.​ DOI: https://doi.org/10.1182/blood.V128.22.1722.1722 

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Authors
Solovey, Maria A.; Wang, Ying; Gattenloehner, Stefan; Hessmann, Elisabeth ; Metzelder, Stephan; Michel, Christian; Ellenrieder, Volker ; Goethert, Joachim R.; Neubauer, Andreas; Burchert, Andreas
Issue Date
2016
Journal
Blood 
ISSN
0006-4971
eISSN
1528-0020
Language
English

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